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The fixed oil and linolenic acid possess significant anti-inflammatory activity against prostaglandin E2, leukotriene and arachidonic acid by virtue of their capacity to block both the lipoxygenase and cyclooxygenase pathways of arachidonic acid metabolism. The oils processed from fresh leaves and seeds of O.
It was noted that Tulsiextracts could significantly reduce the edema when compared with the saline treated control. However, its effect was less than the standard drug.
A study done on rabbits showed that Tulsi has tremendous ability to reduce the oxidative stress produced in the body. A randomized, placebo-controlled, cross over single blind human trial indicated a significant decrease in fasting and postprandial blood glucose levels by Urine glucose levels showed a similar trend.
Further, OS has aldose reductase activity, which may help in reducing the complications of diabetes such as cataract, retinopathy, etc  A study conducted on rats has suggested that constituents of O.
The antipyretic activity was elicited by decreased fever in the rats when the oil was administered topically on their lips. Other clinically important filamentous fungi such as Fusarium solani, P.
This activity is due to the constituents such as methyl chavicol and linalool that are present in the extracts of Tulsi. The fungicidal activity is said to be due to the action of secondary metabolites which are present in Tulsi including alkaloids, glycosides, saponins, tannins, ascorbic acids eugenol and various other metabolites, as mentioned previously.
The effect appears to be due to the antiaggregator action of oil on platelets. This antibacterial activity is thought to be due to the active ingredient eugenol, as mentioned in the text previously. Hence, Tulsi can be used safely even at a higher concentration as compared to sodium hypochorite due to its nonbiofriendly reactions to the developing tooth buds, burning sensation to the tissues, as well as inadvertent allergic reactions. In such studies, microscopically, the cells showed shrunken cytoplasm and condensed nuclei.
The DNA was found to be fragmented when observed in agarose gel electrophoresis. The application of Tulsi extracts in the form of paste has shown promising results in the prevention of DMBA-induced buccal pouch carcinogens.
MNNG is a potent mutagen and induces erosions of the gastric mucosa, an initial precancerous change integral for the initiation of stomach carcinogenesis. Intragastric administration of MNNG induces increased cell proliferation and angiogenesis with evasion of apoptosis leading to well-differentiated squamous cell carcinomas.
A significant decrease in the levels of cytokeratin, CK infiltration , vascular endothelial growth factor, VEGF angiogenesis , proliferating cell nuclear antigen PCNA , glutathione-S-transferase pi key proteins involved in proliferation , and antiapoptotic protein Bcl-2 with simultaneous increase in the proapoptotic proteins Bax, cytochrome c, and caspase 3 were reported.
Tulsi also causes a decrease in the 7,dimethylbenz[a] anthracene-induced genotoxicity, as evaluated by the micronuclei formation in bone marrow cells in mice. These results suggest that, in association with the modulation of the phase I and II detoxification enzymes, Tulsi possesses antigenotoxic effects, and all these might have contributed to the reduction of chemical carcinogenesis.
The heat shock protein, which is altered during carcinogenesis, has also shown a decrease in its concentration.
There was also a concomitant decrease in the phase I enzymes and lipid peroxidation suggesting that O. Two isolated flavonoids vicenin and orientin from O. The combination of O. Multiple studies have shown that luteolin's anticancer property is coupled with the stimulation of apoptosis, inhibition of cell proliferation, angiogenesis, and metastasis. Had met Accord nooit klachten, zyprexa weight gain tulasi tree types ineens kreeg ik hevige hoestbuien, prikkelhoest.
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